One mechanism is to avoid the binding of TAL effectors by mutations of their DNA binding sites, resulting in resistance by loss-of-susceptibility. However, plants have evolved specific strategies to counter the action of TAL effectors and confer resistance. Upon binding to the promoters of plant disease susceptibility genes in a sequence-specific manner, the expression of these host genes is induced. This particular family of type III effectors functions as specific plant transcription factors via a programmable DNA-binding domain. Many plant-pathogenic xanthomonads rely on Transcription Activator-Like (TAL) effectors to colonize their host. 2Biology Department, Universidad Nacional de Colombia, Bogota, Colombia. ![]() ![]() 1UMR IPME, Institut de Recherche Pour le Développement, IRD-CIRAD-Université Montpellier 2, Montpellier, France.Pérez-Quintero 1† Camilo Lopez 1,2 Boris Szurek 1*
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